Myelin dysfunction drives amyloid-β deposition in models of Alzheimer’s disease
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Created: 2nd Jun 2023 at 08:02
Last updated: 13th Jun 2023 at 14:07
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Projects: Default Project - for test purposes, CRC1002: Modulatory Units in Heart Failure, Cardiomyocyte tissues, TRR 274: Checkpoints of Central Nervous System Recovery, SFB 1190: Compartmental Gates and Contact Sites in Cells, MBExC: Multiscale Bioimaging, CRC 1565: Molecular mechanisms and interplay of gene expression processes, CRC 1690: Disease Mechanisms and Functional Restoration of Sensory and Motor Systems, SFB1690 Optogenetics Data - Demo project
Institutions: Default Institution, Universitätsmedizin Göttingen, University of Göttingen
https://orcid.org/0000-0002-9895-2469
Projects: CRC1002: Modulatory Units in Heart Failure, TRR 274: Checkpoints of Central Nervous System Recovery, SFB 1190: Compartmental Gates and Contact Sites in Cells
Web page: Not specified
https://rdp.sfb274.de/ (Research Data Platform) https://gepris.dfg.de/gepris/projekt/408885537 (DFG Gepris entry) The central nervous system (CNS) is a terminally differentiated tissue, where any insult carries a heightened risk - yet the tissue response to these insults is variable and can range from irreversible destruction to almost complete recovery. The rules that instruct these divergent outcomes are still unknown. The aim of this CRC is therefore to understand the biology of the multicellular ...
Programme: Sonderforschungsbereiche/Collaborative Research Centers
Public web page: https://www.sfb274.de/
Submitter: Harald Kusch
Studies: Myelin dysfunction drives amyloid-β deposition in models of Alzheimer’s ...
Assays: No Assays
